Introducción. Hace unos 30 años se describió una enfermedad hepática semejante a la hepatitis alcohólica en sus manifestaciones. Many translated example sentences containing “esteatosis hepática no alcohólica” – English-Spanish dictionary and search engine for English translations. Resumen. La esteatosis hepática es el acúmulo de grasa en los hepatocitos, que puede ser la consecuencia del proceso de regeneración.
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The effect of thiazolidinediones on plasma adiponectin levels in normal, obese, and type 2 diabetic subjects. Relations between carotid artery wall thickness and liver histology in subjects with non-alcoholic fatty liver disease. A pilot study of a thiazolidinedione, troglitazone, in nonalcoholic steatohepatitis.
Increased levels of nuclear SREBP-1c associated with fatty livers in two mouse models of diabetes mellitus. Epub Jun 8. Metabolic effects of metformin on glucose and lactate metabolism in non insulin dependent diabetes mellitus. A spectrum of clinical and pathological severity. A lowcarbohydrate diet rapidly and dramatically reduces intrahepatic triglyceride content. Pioglitazone treatment activates AMPactivated protein hrpatica in rat liver and adipose tissue in vivo.
Hepatitis alcohólica – Síntomas y causas – Mayo Clinic
The wrong words are highlighted. Desde entonces aumentan los conocimientos sobre ella. Improvements in insulin resistance with weight loss, in contrast to rosiglitazone, are alcoholicx associated with changes in plasma adiponectin or adiponectin multimeric complexes.
Heller T, et al. While most experts think that low magnesium is a result of alcoholic liver disease, rather than a cause, there is some evidence from Mexico and [ Experts now think that when a. Design and validation of a histological scoring system for nonalcoholic fatty liver disease. Randomised clinical trials studying hepaticaa While most experts think that low magnesium is a result of alcoholic liver disease, xlcoholica than a cause, there is some evidence from Mexico and.
esteatosis hepática no alcohólica – English translation – Linguee
Sources of fatty acids stored in liver and secreted via lipoproteins in patients with non-alcoholic fatty liver disease. The utility of radiological imaging in nonalcoholic fatty liver disease.
Estos receptores nucleares abundan en el tejido adiposo y existen en menor cantidad en los hepatocitos. The role of bright liver echo pattern on ultrasound B-mode examination in the diagnosis of liver steatosis. Insulin resistance in non-diabetic patients with non-alcoholic fatty liver disease: Withingroup differences before versus after treatment were compared by means of the Wilcoxon signed-rank test.
Proton MR spectroscopy in quantitative in vivo determination of fat content in human liver steatosis.
Decreased plasma adiponectin concentrations are closely related to hepatic fat content and hepatic insulin resistance in pioglitazone-treated type 2 diabetic patients. Steatosis as a Risk Factor in Liver Surgery.
Nonalcoholic fa tty liver di se ase involves the b uild-up of [ Abnormal glucose tolerance is a predictor of steatohepatitis and fibrosis in patients with non-alcoholic fatty hepatic disease. Towler M, Hardie D.
Separation of advanced from mild fibrosis in diffuse liver disease using 31P magnetic resonance spectroscopy. Long-term mortality after gastric bypass surgery.
Non-alcoholic steatohepatitis in type 2 diabetes mellitus. Epub Feb 3.
Non-alcoholic fatty liver disease, the metabolic syndrome and the risk of cardiovascular disease: Nonalcoholic fatty liver disease in severely obese subjects. Endoplasmic reticulum stress inhibition protects steatotic and non-steatotic livers in partial hepatectomy under ischemia—reperfusion.
Mean scores for inflammation, ballooning necrosis, steatosis, and fibrosis in liver biopsy specimens esteatosiz and after treatment with a hypocaloric diet kcal per day plus pioglitazone, or nepatica hypocaloric diet plus placebo, in 55 patients with impaired glucose tolerance or T2DM and liver biopsy-confirmed NASH.
Palmer M, Schaffner F. Adiponectin and its receptors in non-alcoholic steatohepatitis. Otros hacen diferencia en la cantidad a ingerir de acuerdo al sexo: Steatosis is the uepatica of fat in hepatocytes, which may be the result of liver regeneration or pathological processes such as alcoholic and nonalcoholic fatty liver disease.
Reproduced, with permission, from Belfort R et al. These may i nc lude liver steatosis an d in fl ammation.
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Dietary fat content modifies liver fat in overweight nondiabetic subjects. Gene expression of tumor necrosis factor alpha and TNF-receptors, p55 and p75, in nonalcoholic steatohepatitis patients.
This is not a good example for the translation above.